A reduction in load and therefore firing of sensitised structures (not necessarily damaged) will also affect the pain experience. Sensory signalling from the periphery is also heavily involved in the pain experience. There could be several possible mechanisms for how Dynamic Taping can influence pain. may reduce pain and some research on the hip is showing preliminary support for this. Directly modifying load by creating some of the force externally or indirectly modifying it by changing movement patterns, reducing compression etc. In many cases however the pain present is still load dependent. Do we need to have a more comprehensive preparatory phase and education? Can we use Dynamic Tape to normalise movement and to challenge faulty beliefs about pain, movement and damage? As clinicians we need to determine if there is structural damage, is there nociception or inflammatory influences, is there peripheral sensitisation and primary hyperalgesia? Perhaps there is no ongoing damage, nociception or inflammation but central sensitisation is continuing to wind up the pain perhaps from non nociceptive peripheral drivers (possibly coming from maladaptive movement patterns). That does not mean however that we should forget about the tissues or structure entirely. Beliefs, expectations, past experiences, social and environmental factors all influence the pain experience. Pain can be present as a warning sign before any damage has occurred. Pain can persist long after structural damage is thought to have repaired. Pain is complex and is not well correlated with tissue damage.
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